Poststreptococcal glomerulonephritis is a kidney disease prevalent in third world countries with a frequency between "9.5 and 28.5 new cases per 100,000 individuals per year" (Rodriguez-Iturbe et al., 2008). Poststreptococcal glomerulonephritis is considered an immune complex disease because glomerular damage results from interactions between antigen-antibody complexes and the complement system. Potential antigens that form the antigen-antibody complex are nephritic plasmin-binding protein, nephritic strain-associated protein, and endostreptosin (Stickler, 2003). Pathophysiology The lesion theory clarifies the process by which streptococcal antigen reacts with a host antibody, forming a soluble complex. which is then deposited at a sub-endothelial level along the basement membrane, reacting with the complement system. The complement cascade then encourages phagocytosis and digestive activity at the basement membrane, which directs fragmentation and disruption subsequently leading to characteristic outcomes for a patient with poststreptococcal glomerulonephritis (Stickler, 2003)....